It's a great study. I would welcome if they did the same experiment and analysis on groups of people who had an anxiety disorder diagnosed and got healed, preferrably with CBT or other non-pharmacological methods alone, and a healthy population. It would be interesting to see the effects of therapy on this mechanism and whether an higher excitable FPI stays activated in the healed cohort or it's exitability is less, or the amygdala activation is again the root of all of this. It's notable that some healthy individuals had high FPI excitability but were not anxious.